Research paperArterial injury-induced smooth muscle cell proliferation in rats is accompanied by increase in polyamine synthesis and level
References (21)
Growth factors and the pathogenesis of atherosclerosis
Atherosclerosis
(1986)- et al.
Induction of ornithine decarboxylase activity and putrescine synthesis in arterial smooth muscle cells stimulated with platelet-derived growth factor
Exp. Cell Res.
(1987) - et al.
The relationship between levels and rates of synthesis of polyamines during mammalian cell cycle
Life Sci.
(1981) A rapid and sensitive method for quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
Anal. Biochem.
(1976)- et al.
Protein measurement with the Folin phenol reagent
J. Biol. Chem.
(1951) - et al.
The role of endothelial injury and platelet and macrophage interactions in atherosclerosis
Circulation
(1984) - et al.
Mechanisms of stenosis after arterial injury
Lab. Invest.
(1983) - et al.
Kinetics of cellular proliferation after arterial injury. I. Smooth muscle growth in the absence of endothelium
Lab. Invest.
(1983) - et al.
Significance of quiescent smooth muscle migration in the injured rat carotid artery
Circ. Res.
(1985) - et al.
Vascular smooth muscle cell growth kinetics in vivo in aged rats
There are more references available in the full text version of this article.
Cited by (26)
Local inhibition of ornithine decarboxylase reduces vascular stenosis in a murine model of carotid injury
2013, International Journal of CardiologyCitation Excerpt :Considering the multiplicity of phenomena in which polyamines are involved, they could play a key role in restenosis progression, as well as to show a potential as therapeutic targets to prevent or reduce this recurrent pathophysiological phenomenon. Indirect evidence for a role of polyamines in stenosis progression has come from preclinical models of vascular injury [5–8]. These findings further supported a role for growth- and migration-stimulatory polyamines in stenosis progression.
Lysophosphatidylcholine regulates cationic amino acid transport and metabolism in vascular smooth muscle cells. Role in biosynthesis
1997, Journal of Biological ChemistryCharacterization of Polyamine Transport in Rat Aortic Smooth Muscle Cells
1994, Journal of Surgical ResearchPolyamines in testosterone-induced hypertrophic and antifolate-induced hyperplastic mouse kidney. Differential effect of α-difluoromethylornithine
1993, BBA - Molecular Basis of DiseaseArterial response to mechanical injury: balloon catheter de-endothelialization
1992, Atherosclerosis
Copyright © 1990 Published by Elsevier Ltd.