The incidence of abnormal pattern electroretinography in optic nerve demyelination
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Electrophysiology in neuro-ophthalmology
2021, Handbook of Clinical NeurologyCitation Excerpt :Ischemic optic neuropathy often results in PERG N95 reduction (Atilla et al., 2006; Parisi et al., 2008) associated with loss of the retinal ganglion cells (Almarcegui et al., 2001). The PERG P50 component is more frequently affected in ischemic optic neuropathy than in optic neuritis (Holder, 1991a,b; Almarcegui et al., 2001), reflecting the severity of retinal ganglion cell involvement. Compressive lesions of the anterior visual pathway cause insidious and progressive visual impairment with visual field defects that reflect the location of the lesion in the visual pathway.
Application of electrophysiological tests in the study of neurological diseases with visual involvement
2012, Archivos de la Sociedad Espanola de OftalmologiaInternational federation of clinical neurophysiology: Recommendations for visual system testing
2010, Clinical NeurophysiologyCitation Excerpt :In the absence of visual symptoms, and in the presence of normal visual acuity, the presence of a delayed PR-VEP can be taken as evidence of optic nerve disease; there is no need under such circumstances to confirm normal macular function. PERG has a normal P50 component in the majority of patients with optic nerve demyelination; approximately 40% will have PERG N95 component abnormalities (Holder, 1991). The VEP can also be abnormal in a variety of degenerative diseases of the nervous system such as Friedreich ataxia, other hereditary ataxias, familial spastic paraplegia, adrenoleukodystrophy (ALD), or hereditary motor and sensory neuropathies.
Correlation between functional and structural assessments of the optic nerve and retina in multiple sclerosis patients
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