The effect of oxaloacetate ACTH and cortisol on the liver glycogen levels of Tilapia mossambica

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Abstract

  • 1.

    1. A single injection of oxaloacetate into Tilapia mossambica caused a rapid depletion of liver glycogen. A series of six injections given over a 72-hr period kept liver glycogen levels low and caused a marked increase in a liver size.

  • 2.

    2. After removal of oxaloacetate, liver weight continued to increase for about 36 hr and then decreased. Liver glycogen rose steadily for 108 hr, and then fell sharply. A net synthesis of liver glycogen was observed.

  • 3.

    3. Two injections of ACTH, or six injections of cortisol, after oxaloacetate, served to stimulate liver glycogen synthesis.

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Supported by NSF GB-7200 and was supported by Biomedical Support Grant Fr-07053 from the General Research Support Branch, Division of Research Resources, Bureau of Health Professions Education and Manpower Training, National Institutes of Health.

Supported by a NASA Pre-doctoral Fellowship. Present address: Biology Department, University of Houston, Houston, Texas 77004.

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