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CALR loss-of-heterozygosity as a potential driver for extramedullary AML

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Acknowledgements

A.P.A.T. is supported by the Professor Dr. Max Cloëtta Foundation.

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Author notes

  1. Eugenia Haralambieva

    Present address: Institute for Clinical Pathology Medica, Hottingerstrasse 9/11, 8032, Zurich, Switzerland

Authors and Affiliations

  1. Department of Medical Oncology and Hematology, University Hospital Zurich and University of Zurich, Rämistrasse 100, 8091, Zurich, Switzerland

    Marie-Christine Weller, Alexandre Pierre André Theocharides & Stefan Balabanov

  2. Department of Pathology and Molecular Pathology, University Hospital Zurich and University of Zurich, Zurich, Switzerland

    Eugenia Haralambieva & Marco Matteo Bühler

  3. Division of Hematology and Oncology, Kantonsspital Muensterlingen, Muensterlingen, Switzerland

    Rudolf Benz

Authors
  1. Marie-Christine Weller
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  2. Eugenia Haralambieva
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  3. Marco Matteo Bühler
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  4. Rudolf Benz
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  5. Alexandre Pierre André Theocharides
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  6. Stefan Balabanov
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Contributions

M.C.W., E.H., M.M.B., R.B., A.P.A.T., and S.B. provided and analyzed the data; M.C.W., A.P.A.T., and S.B. wrote the paper. S.B. and A.P.A.T. contributed equally to this work.

Corresponding author

Correspondence to Stefan Balabanov.

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The authors declare no competing interests.

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Weller, MC., Haralambieva, E., Bühler, M.M. et al. CALR loss-of-heterozygosity as a potential driver for extramedullary AML. Ann Hematol 101, 2571–2573 (2022). https://doi.org/10.1007/s00277-022-04976-8

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  • DOI: https://doi.org/10.1007/s00277-022-04976-8

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