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Trans-activation by hepatitis B virus X protein is mediated via a tumour promoter pathway

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Chronically Evolving Viral Hepatitis

Part of the book series: Archives of Virology ((ARCHIVES SUPPL,volume 4))

Summary

We have studied the c-myc gene as a possible target of HBV X protein in liver carcinogenesis. Our results indicate that trans-activation by X protein occurs via PKC/AP1 signal transduction, suggesting a possible two-step mechanism in HBV related liver carcinogenesis.

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Authors and Affiliations

  1. Max-Planck-Institut für Biochemie, D-8033, Martinsried, Federal Republic of Germany

    Dr. A. S. Kekulé, U. Lauer, L. Weiß, P. H. Hofschneider & R. Koshy

Authors
  1. Dr. A. S. Kekulé
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  2. U. Lauer
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  3. L. Weiß
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  4. P. H. Hofschneider
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  5. R. Koshy
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Editor information

Editors and Affiliations

  1. Titolare II Cattedra Clinica, Malattie Tropicli e Infettive, Policlinico Umberto, Roma, Italy

    C. De Bac  & G. Taliani  & 

  2. Institut für Medizinische Virologie, Justus-Leibig-Universität, Giessen, Federal Republic of Germany

    W. H. Gerlich

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© 1992 Springer-Verlag

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Cite this paper

Kekulé, A.S., Lauer, U., Weiß, L., Hofschneider, P.H., Koshy, R. (1992). Trans-activation by hepatitis B virus X protein is mediated via a tumour promoter pathway. In: De Bac, C., Taliani, G., Gerlich, W.H. (eds) Chronically Evolving Viral Hepatitis. Archives of Virology, vol 4. Springer, Vienna. https://doi.org/10.1007/978-3-7091-5633-9_13

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  • DOI: https://doi.org/10.1007/978-3-7091-5633-9_13

  • Publisher Name: Springer, Vienna

  • Print ISBN: 978-3-211-82350-7

  • Online ISBN: 978-3-7091-5633-9

  • eBook Packages: Springer Book Archive

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