Abstract
Acute pancreatitis is a common disorder the incidence of which is increasing in emergency units and which is still associated with high mortality.
The main pathophysiological event that characterises acute pancreatitis consists in the positive feedback of auto-digestive activation of pancreatic pro-enyzmes within the gland along with the systemic extension of the inflammatory process.
This is a preview of subscription content, log in via an institution.
Buying options
Tax calculation will be finalised at checkout
Purchases are for personal use only
Learn about institutional subscriptionsPreview
Unable to display preview. Download preview PDF.
References
Noronha M, Batista A, Bordalo O (1984) Pancreatitis, concepts and classification. Gyr K, Singer M and Sarles H. Excerpta Medica, Amsterdam, New York, Oxford 61–65
Lerch MM and Adler G. Acute pancreatitis — recent developments and perspectivey. Urban und Schwarzenberg 64–69
Ranson JHC, Rifkind KM, Roses DF et al (1974) Prognostic signs and the role of operative management in acute pancreatitis. Surg Gynecol Obstet. 139: 69–81
Blainey SL, Imrie CW, O’Neill J et al (1984) Prognostic factors in acute pancreatitis. Gut 25: 1340–46
Larvin M, McMahon MJ (1989) APACHE II score for assessment and monitoring of acute pancreatitis. Lancet 2: 201–5
Domínguez-Muñoz JE, Carballo F, Garcia MJ et al (1991) Clinical usefulness of polymorphonuclear elastase in predicting the severity of acute pancreatitis: results of a multicenter study. Br J Surg 78: 1230–4
Viedma JA, Pérez-Mateo M, Domínguez-Muñoz JE et al (1992) Role of interleukin-6 in acute pancreatitis: comparison with C-reactive protein and phospholipase A. Gut 33: 1264–7
Gross V, Schölmerich J, Leser HG et al (1990) Granulocyte elastase in assessment of severity of acute pancreatitis: comparison with acute phase proteins C-reactive protein, α1-antitrypsin, and protease inhibitor α2-macroglobulin. Dig Dis Sci 35: 97–105
Domínguez-Muñoz JE, Malfertheiner P (1993) Management of acute pancreatitis. The Gastroenterologist 1: 248–256
Klöppel G, Maillet B (1991) Chronic pancreatitis. Evolution of the disease. Hepato-Gastroenterology 38: 408–412
Dreiling DA, Bordalo O (1977) A toxic-metabolic hypothesis of pathogenesis of alcoholic pancreatitis. Alcoholism Clin Exp Res 1: 293–299
Noronha M, Salgadinho A, Ferreira De Almeida MJ, Dreiling DA and Bordalo O (1981) Alcohol and the pancreas. I Clinical association and histopathology of minimal pancreatic inflammation. Am J Gastroenterol 76: 114–119
Noronha M, Bordalo O, Dreiling DA (1981) Alcohol and the pancreas. II Pancreatic morphology of advanced alcoholic pancreatitis. Am J Gastroenterol 76: 120–124
Guyan PM, Uden S, Braganza JM (1990) Heightened free radical activity in pancreatitis. Free Radical Biology & Medicine 8: 347–354
Cameron JL, Zuidema GD, Margolis S (1975) A pathogenesis for alcoholic pancreatitis. Surgery 77: 754–763
Noronha M, Dreiling DA, Bordalo MD (1983) Sequential changes from minimal pancreatic inflammation to advanced alcoholic pancreatitis. Z Gastroenterologie 21: 666–673
Editor information
Editors and Affiliations
Rights and permissions
Copyright information
© 1997 Springer-Verlag Berlin Heidelberg
About this paper
Cite this paper
Bordalo, O.R., Palma, R.F. (1997). Clinical Presentation and Course of Acute Pancreatitis. In: Malfertheiner, P., Domínguez-Muñoz, J.E., Schulz, HU., Lippert, H. (eds) Diagnostic Procedures in Pancreatic Disease. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-60580-2_3
Download citation
DOI: https://doi.org/10.1007/978-3-642-60580-2_3
Publisher Name: Springer, Berlin, Heidelberg
Print ISBN: 978-3-642-64464-1
Online ISBN: 978-3-642-60580-2
eBook Packages: Springer Book Archive