Journal of Biological Chemistry
Volume 293, Issue 1, 5 January 2018, Pages 226-244
Molecular Bases of DiseaseReceptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairmentRAGE mediates neurodegeneration in sepsis
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amyloid-β (Aβ)
neurodegeneration
neuroinflammation
receptor for advanced glycation end products (RAGE)
sepsis
Tau protein (Tau)
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This work was supported by grants from Conselho Nacional de Desenvolvimento Cientifico e Tecnologico (to D. P. G., J. C. F. M., J. Q., and F. D.-P.), FundaçΔo de Amparo à Pesquisa do Estado do Rio Grande do Sul (to D. P. G. and J. C. F. M.), FundaçΔo de Amparo à Pesquisa e InovaçΔo do Estado de Santa Catarina (to J. Q. and F. D.-P.), Propesq/UFRGS (to D. P.G. and J. C. F. M.), FundaçΔo CAPES, Instituto Cérebro e Mente (to J. Q.) and UNESC (to T. B., J. Q., and F. D.-P.). The authors declare that they have no conflicts of interest with the contents of this article.
© 2018 by The American Society for Biochemistry and Molecular Biology, Inc.