Journal of Biological Chemistry
Volume 273, Issue 37, 11 September 1998, Pages 23884-23891
CELL BIOLOGY AND METABOLISMA Hyperprostaglandin E Syndrome Mutation in Kir1.1 (Renal Outer Medullary Potassium) Channels Reveals a Crucial Residue for Channel Function in Kir1.3 Channels*
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This work was funded in part by Grants Ka1175/1-2, Da177/7-2, and S.E.263/15-1 of the Deutsche Forschungsgemeinschaft and a grant of the Karl und Lore Klein-Stiftung (to R. P. M.).The costs of publication of this article were defrayed in part by the payment of page charges. The article must therefore be hereby marked “advertisement” in accordance with 18 U.S.C. Section 1734 solely to indicate this fact.
The nucleotide sequence(s) reported in this paper has been submitted to the GenBank™/EMBL Data Bank with accession number(s) AS049076.
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These authors contributed equally to this work.
© 1998 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology.