LetterRe: Estimation of Bias in Nongenetic Observational Studies Using “Mendelian Triangulation” by Bautista et al.
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Genetic determinants of complement activation in the general population
2024, Cell ReportsMendelian randomization analysis of the causal impact of body mass index and waist-hip ratio on rates of hospital admission
2022, Economics and Human BiologyCitation Excerpt :As the hospital admission count variance (55.3) was greater than the mean hospital admission count (1.89), we used a quasi-Poisson model instead (Ver Hoef and Boveng, 2007). Standard errors for the causal IV estimates were estimated using Taylor series expansions (Thomas, Lawlor, and Thompson, 2007). Weighted genetic risk scores (GRS)s were constructed for BMI (76 SNPs) (Locke et al., 2015), WHR (39 SNPs) (Shungin et al., 2015) and WHRadjBMI (48 SNPs) (Shungin et al., 2015).
Associations between plasma fatty acid concentrations and schizophrenia: a two-sample Mendelian randomisation study
2021, The Lancet PsychiatryCitation Excerpt :Details are available in the appendix (pp 1–2). For the FADS gene cluster and ELOVL2 gene single SNP analyses, ratio estimates were calculated by dividing the SNP-schizophrenia effect estimate by the SNP-fatty acid effect estimate with SEs derived using the first term from a delta method expansion for the ratio estimate.25 We also did a multivariable Mendelian randomisation26 to simultaneously estimate the direct effects of omega-3 and omega-6 fatty acid concentrations on schizophrenia.
Lipid and metabolic syndrome traits in coronary artery disease: A Mendelian randomization study
2021, Journal of Lipid ResearchCannabis use and atherosclerotic cardiovascular disease: a Mendelian randomization study
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