Elsevier

Methods in Enzymology

Volume 375, 2003, Pages 297-322
Methods in Enzymology

HMGA Proteins: Isolation, Biochemical Modifications, and Nucleosome Interactions

https://doi.org/10.1016/S0076-6879(03)75020-4Get rights and content

Publisher Summary

High mobility group A (HMGA) proteins act as architectural transcription factors that function by recognizing and modulating the structure of both DNA and chromatin substrates. A number of chromatographic procedures have been successfully used to purify HMGA proteins eluted by either the salt or acid extraction method, ion-pair reversed-phase high-performance liquid chromatography (RP-HPLC) is employed for such separations because it provides a fast and convenient method for obtaining highly purified forms of each of the different members of the HMGA protein family. Acid extracted proteins isolated from mammalian cells growing in culture, RP-HPLC chromatography can be used to separate the HMGA1a and HMGA1b isoform proteins not only from each other but also from other contaminating protein species in the extracts. A variety of procedures have been employed to determine the sites of individual types of in vivo biochemical modifications on HMGA proteins, mass spectrometry (MS) techniques have proved to be the most efficient and facile methods for analyzing the complex patterns of multiple modifications that are simultaneously present on these proteins in living cells. Matrix-assisted laser desorption/ionization time-of-flight (MALDI/ TOF) MS has been successfully used in the laboratory to analyze the types and in some cases even the actual locations, of posttranslational modifications (PTMs) on HMGA1 proteins32, 42 isolated from mammalian cells. Methods for analyzing the modes of interaction of HMGA1 proteins with both native and in vitro reconstituted nucleosome core particles are also presented.

Introduction

The mammalian HMGA family of high mobility group (HMG) nonhistone proteins [formerly called HMG-I(Y) proteins1] has been referred to as a “hub” of nuclear function because of its intimate involvement in a wide variety of biology processes.2 An extensive literature suggests that HMGA proteins act as architectural transcription factors that function by recognizing and modulating the structure of both DNA and chromatin substrates and, as a consequence, have the ability to participate in a diverse array of nuclear events ranging from DNA replication and repair to the regulation of gene transcription and mRNA splicing and the control of integration of retroviruses into the genome (reviewed in Ref. 3). HMGA proteins have also attracted considerable medical interest because their aberrant expression or overexpression has been demonstrated to lead to neoplastic transformation of cells and to promote metastatic progression of cancers (reviewed in Refs. 3, 4, 5, 6).

Section snippets

HMGA Proteins Exhibit Unusual Properties

In both mice and humans, the principal members of the HMGA protein family (i.e., HMGA1a, HMGA1b, and HMG2) are produced by translation of alternatively spliced mRNA transcripts encoded by two different genes, HMGA1 and HMGA2 (reviewed in Refs. 6, 7). HMGA proteins share a number of common biochemical, biophysical, and biological properties that distinguish them from other nuclear proteins. The major HMGA protein species are small (∼10.6–12 kDa), have unusually high concentrations of basic,

Production and Purification of Recombinant HMGA Proteins

Wild-type, mutant, and truncated forms of recombinant HMGA proteins produced in either bacteria or insect cells have been widely used in studies to elucidate their in vitro biochemical, biophysical, and biological characteristics. Unfortunately, however, it is often difficult to isolate appreciable quantities of recombinant HMGA proteins because their levels of production in many host cell expression systems are frequently low. Among the factors that contribute to this low level of expression

Isolation and Purification of HMGA Proteins from Mammalian Cells and Tissues

Two different methods have been widely used to extract HMGA proteins from the nuclei and chromatin of mammalian cells. Either they can be eluted by extraction with dilute acids, as described above for recombinant proteins, or they can be extracted with a buffered solution containing 0.3–0.4 M NaCl.27 There are advantages and disadvantages to both methods. Although salt extraction is mild and allows for relatively efficient recovery of native, nondenatured HMGA proteins, it results in the

In Vivo Biochemical Modifications of HMGA Proteins

HMGA proteins isolated from living cells exhibit complex patterns of postsynthetic phosphorylations, acetylations, and methylations, making them among the most highly modified proteins in the mammalian nucleus (reviewed in Refs. 2, 3). These modifications are dynamic and rapidly change as a function of the cell cycle,30, 31 the state of cellular differentiation,32 in response to intra- and extracellular signaling events,32 and during the activation of apoptotic cell death.33, 34

The diagram in

Determination of In Vivo Biochemical Modifications by Mass Spectrometry

Although a variety of procedures have been employed to determine the sites of individual types of in vivo biochemical modifications on HMGA proteins, mass spectrometry (MS) techniques have proved to be the most efficient and facile methods for analyzing the complex patterns of multiple modifications that are simultaneously present on these proteins in living cells. Matrix-assisted laser desorption⧸ionization time-of-flight (MALDI/TOF) MS has been successfully used in our laboratory to analyze

HMGA Proteins Bind to Nucleosome Core Particles Both In Vitro and In Vivo

Although HMGA proteins have received a considerable amount of attention because of their active role in regulating the transcription of more than 40 different eukaryotic and viral genes by participating in the formation of stereospecific, multiprotein complexes called “enhanceosomes” on their promoter⧸enhancer regions (reviewed in Refs. 3, 48), these proteins have long been recognized as being important participants in many other nuclear processes. HMGA proteins are integral components of both

HMGA1 Binding to Nucleosomes Is Orientation Specific

Interaction of HMGA1 proteins with nucleosomes not only induces localized changes in the rotational setting of DNA on core particles,13 it also binds to A⧸T-rich stretches of DNA on the nucleosome surface in a orientation-specific manner.53 HMGA1 proteins participate in the transcriptional induction of the gene encoding the α subunit of the human interleukin 2 receptor (IL-2Rα) in stimulated T lymphocytes by controlling the formation of an enhanceosome on the gene's 5′ proximal promoter region.

Isolation, Mutagenesis, and Radiolabeling of DNA Fragments

A cloned 581-bp BamHI–PstI restriction fragment encompassing nucleotides −472 to +109 of the human IL-2Rα gene and its 5′ proximal promoter region57 serves as the starting material for isolating subfragments of the promoter containing the PRRII element for use in in vitro chromatin reconstitution experiments. Standard polymerase chain reaction (PCR) techniques22 are used to amplify a 277-bp promoter fragment that encompasses PRRII and its flanking regions, using the following PCR primer pair:

A⧸T-Hook Peptide Motif Binds Nucleosome Core Particles

Studies of cocomplexes of HMGA1 proteins with synthetic DNA substrates, using solution nuclear magnetic resonance (NMR) techniques, revealed that A⧸T-hook peptides that interact with the minor groove of A⧸T-rich sequences of DNA do so in a direction-specific manner,8 raising a question concerning whether the A⧸T-hook regions of these proteins are also responsible for mediating the directional interactions with nucleosome core particles. Of even greater biological importance is determining

A⧸T Hooks Are Essential Components of Many Chromatin-Remodeling Complexes

The observation that HMGA proteins bind to nucleosome core particles via their A⧸T-hook motifs and, thereby, induce localized changes in the structure of DNA on the surface of core particles has taken on unanticipated significance. It is now becoming apparent that A⧸T hook-containing proteins are essential components of many of the multi-protein, ATP-dependent chromatin-remodeling complexes (or “machines”; CRMs) found in yeast, Drosophila, and mammalian cells (reviewed in Refs. 2, 3). For

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