Sino-atrial disease: A report on 13 cases
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Cited by (33)
Sinus Node Dysfunction
2023, Cardiology ClinicsSinus Node Dysfunction
2021, Cardiac Electrophysiology ClinicsCitation Excerpt :Furthermore, SND diagnostic criteria are not consistent among published reports, and distinguishing intrinsic SND from extrinsic causes during the review of individual medical records is impractical, thereby undermining the value of epidemiologic estimates. In an early study, Kulbertus and colleagues50 estimated that in persons older than 50 years attending a cardiology clinic, the incidence of symptomatic SND is approximately 5 in 3000 (about 0.17%). More recently, using access to much larger study populations (Atherosclerotic Risk in Communities study [ARIC] and Cardiovascular Health Study [CHS]), Jensen and colleagues51 estimated the development of SND in 20,572 subjects (mean age 59 years) who were deemed free of SND findings at entry.
The Anatomical Substrate for Sick Sinus Syndrome in Dogs
2021, Journal of Comparative PathologyCitation Excerpt :The mechanism responsible for atrial tachyarrhythmias involves re-entrant excitation or spontaneous automaticity of an ectopic atrial focus (Cote, 2010). The fibrotic lesions in the atrial wall can constitute anatomical obstacles to conduction, which should offer ideal paths for re-entrant excitation (Kulbertus and de Leval-Rutten, 1973; Demoulin and Kulbertus, 1978). In addition, the loss of pacemaker function due to depletion of nodal cells would account for the predisposition of the injured atrial myocardium to delayed and non-homogeneous conduction (Monfredi and Boyett, 2015).
Indications for permanent pacing in dogs and cats
2019, Journal of Veterinary CardiologyCitation Excerpt :In human beings, involvement of the right atrial wall has been proven when conspicuous alterations of the SA node and its approaches have been documented. These changes include total or subtotal destruction of the SA node, areas of nodal-atrial discontinuity, inflammatory or degenerative changes in the nerves and ganglias surrounding the SA node, and pathological changes in the atrial wall [18,57–61]. It has also been proven that, although frequently associated with underlying heart disease and seen most often in the elderly, SSS may occur in the fetus, infant, and child without apparent cause [62].
The Anatomical Basis of Bradycardia-Tachycardia Syndrome in Elderly Dogs with Chronic Degenerative Valvular Disease
2012, Journal of Comparative PathologyCitation Excerpt :The similarity of the SA nodal lesions after various durations of artificial pacing suggests that these histological alterations are the cause of BTS. Although the aetiology of the extensive damage to the SA node (i.e. depletion of the nodal cells with a corresponding increase of fibrous or fibro-fatty tissue) is not clear, it is interesting to note that qualitatively the pathological changes resembled those associated with ageing in human patients with atrial arrhythmias (Kaplan, et al., 1973; Kulbertus, et al., 1973; Thery et al., 1977; Sugiura and Ohkawa, 1980; Bharati, 2001). Thus, it is possible that the pathological process in the SA node of the dogs was also fundamentally a phenomenon related to accelerated ageing.
Cardiovascular disease in elderly patients
1996, Mayo Clinic Proceedings
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Fonds National de la Recherche Scientifique Belge, Division of Cardiology, Department of Medical Clinics and Semiology, University of Liege School of Medicine, Liege.
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Division of Cardiology, Department of Medical Clinics and Semiology, University of Liege School of Medicine.
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Department of Morbid Anatomy, University of Liege School of Medicine, Liege.