Effect of Valsalva maneuver on surface electrocardiographic P-wave dispersion in paroxysmal atrial fibrillation
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2015, Journal of CardiologyCitation Excerpt :The activation of the RAAS causes atrial fibrosis, cardiomyocyte apoptosis, and elicits reactive oxygen species generation [77]. Furthermore, the sympathetic nervous system is overactive in insulin-resistant states [78], resulting in a significant prolongation of P-wave duration and PWD [79,80]. In addition, insulin resistance promotes activation of many proinflammatory transcription factors [81] and induces the generation of reactive oxygen species [82,83].
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2019, Journal of ElectrocardiologyCitation Excerpt :The lack of difference in Valsalva ratio, RMSSD, and SDNN between migraineurs and controls, suggest unchanged autonomic cardiovascular reflex in spite of the overall autonomic dysregulation in migraine. The Valsalva ratio is derived by the maximum heart rate by the minimum heart rate after the Valsalva maneuver, and reflects the parasympathetic and sympathetic interaction in baroreflex in response to blood pressure changes [33,34]. RMSSD and SDNN are indicators for primarily vagal-mediated changes in RR intervals [6].