Senile dementia of Lewy body type: A clinically and neuropathologically distinct form of Lewy body dementia in the elderly
References (52)
- et al.
Pathological changes in the nucleus of Meynert in Alzheimer's and Parkinson's diseases
J. Neurol. Sci.
(1983) - et al.
Diffuse Lewy body disease
Lancet
(1989) - et al.
Extensive loss of choline acetyltransferase activity is not reflected by neuronal loss in the nucleus of Meynert in Alzheimer's disease
Neurosci. Lett.
(1982) - et al.
Clinically and pathologically distinct form of dementia in the elderly
Lancet
(1989) - et al.
Observations on the brains of non-demented old people
J. Neurol. Sci.
(1968) - et al.
Observations on the brains of demented old people
J. Neurol. Sci.
(1970) - et al.
A comparison of Degenerations in Cerebral Cortex and Brainstem
Parkinson's disease: clinical and etiopathology
- et al.
Psychogeriatric ascertainment and assessment for treatment within an acute medical ward setting
Age Ageing
(1974) - et al.
The association between quantitative measures of dementia and of senile change in the cerebral grey matter of elderly subjects
Br. J. Psychiat.
(1968)
Mental status of patients with parkinson disease
J. Clin. Neuropsychol.
Parkinson disease, dementia, and Alzheimer disease: clinicopathological correlations
Ann. Neurol.
Neurological Anatomy in Relation to Clinical Medicine
Pathologic correlates of dementia in Parkinson's disease
Arch. Neurol. (Chic.)
Demonstration of neurofibrillary tangles in paraffin sections: a quick and simple method using a modification of Palmgren's method
Med. Lab. Sci.
Neuropathologic and clinical features of Parkinson's disease in Alzheimer's disease patients
Neurology
Cholinergic-dependent cognitive deficits in Parkinson's disease
Ann. Neurol.
Presenile dementia with Lewy bodies and neurofibrillary tangles
Arch. Neurol. (Chic.)
Dementia in idiopathic Parkinson's disease: a neuropathological study on 32 cases
Acta Neuropathol. (Berl.)
Dementia in Parkinson's disease
Br. J. Psychiat.
The progression of idiopathic Parkinson's disease is not explained by age-related changes
Clinical and pathological comparisons with post-encephalitic parkinsonian syndrome
Acta Neuropathol. (Berl.)
Clinical and pathological features of diffuse cortical Lewy body disease (Lewy body dementia)
Brain
Cerebral blood flow in dementia
Arch. Neurol. (Chic.)
Dementia in Parkinson's disease: a neuropathological study
Neurology
Dementia in Parkinson's disease
Arch. Neurol. (Chic.)
Cited by (568)
Dementia: A journey from cause to cure
2023, Nanomedicine-Based Approaches for the Treatment of DementiaLocus coeruleus neurons are most sensitive to chronic neuroinflammation-induced neurodegeneration
2020, Brain, Behavior, and ImmunityCitation Excerpt :Progressive pathological staging and degeneration in Parkinson’s disease (PD) occur along a caudo-rostral axis, especially for patients with young onset and long duration of the disease (Braak et al., 2003; Rietdijk et al., 2017). Noradrenergic locus coeruleus (NE-LC) neurons are among the earliest and most severe sites of degeneration in PD (McKee et al., 2013; Perry et al., 1990; Zarow et al., 2003). Specifically, NE-LC neurons are affected as early as at stage 2 during PD progression, while nigral dopaminergic (DA) neurons and hippocampal/cortical neurons are affected at stage 3 and 5, respectively (Braak et al., 2003).
Distinguishing between dementia with Lewy bodies and Alzheimer's disease using metabolic patterns
2020, Neurobiology of AgingCitation Excerpt :Dementia with Lewy bodies (DLB) is the second most common cause of degenerative dementia after Alzheimer's disease (AD) (Hansen et al., 1990; Perry et al., 1990).
Cell-free amplification of prions: Where do we stand?
2020, Progress in Molecular Biology and Translational ScienceCitation Excerpt :Even the phenotypical heterogeneity that can be found within each group of diseases is thought to be associated with minimal variations in the structure of the protein. For instance, different structural variants of αS can be responsible for different pathological phenotypes of PD,92,93 DLB94,95 or even MSA. This latter might be further classified in parkinsonian (MSA-P) and cerebellar (MSA-C) phenotype, thus supporting a possible contribution of different αS strains.96