Appropriateness of DSM-III-R criteria for posttraumatic stress disorder
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Cited by (113)
Young Women's Experiences Obtaining Judicial Bypass for Abortion in Texas
2019, Journal of Adolescent HealthThe class-dimensional structure of PTSD before and after deployment to Iraq: Evidence from direct comparison of dimensional, categorical, and hybrid models
2016, Journal of Anxiety DisordersCitation Excerpt :Empirically, identification and inclusion of a necessary etiological criterion for a mental disorder such as PTSD, requires a clearly defined pathogenic pathway between the causative factor and the onset and maintenance of the disorder. Although exposure to a traumatic event is widely accepted to be a specific etiological factor in the development of PTSD, endorsement of PTSD symptoms occurs in the absence of exposure to traumatic events (Bodkin, Pope, Detke, & Hudson, 2007; Erwin, Heimberg, Marx, & Franklin, 2006; Gold, Marx, Soler-Baillo, & Sloan, 2005; Simons & Silveira, 1994; Solomon & Canino, 1990). Given plethora findings suggesting PTSD symptoms are common among non-traumatized populations, the assumption that the disorder is a discrete condition arising following a traumatic event requires further testing.
Posttraumatic stress disorder in medical-legal practice
2013, Revista Espanola de Medicina LegalPosttraumatic stress symptom clusters associations with psychopathology and functional impairment
2010, Journal of Anxiety DisordersCitation Excerpt :This has led several authors to assert that the avoidance/numbing symptom cluster drives the diagnosis of PTSD (Breslau, 2001; Norris et al., 2002; North et al., 2009). The relatively crucial importance of Criterion C may be due to at least two reasons, Criterion C symptoms being less prevalent than Criteria B and D symptoms in traumatized populations (Ehlers et al., 1998; Foa, Riggs, & Gershuny, 1995; North et al., 1999; Solomon & Canino, 1990), and\or it may be due to the higher number of Criterion C symptoms required. Thus, the findings that Criterion C predicts PTSD (Maes et al., 1998; North et al., 1999; North et al., 2005) as well as the conclusion that “group C is a marker for PTSD” (North et al., 2009, p. 38) may be a consequence of the current conceptualization of PTSD rather than the importance of the avoidance/numbing symptoms themselves.
A multidimensional spectrum approach to post-traumatic stress disorder: comparison between the Structured Clinical Interview for Trauma and Loss Spectrum (SCI-TALS) and the Self-Report instrument (TALS-SR)
2009, Comprehensive PsychiatryCitation Excerpt :Recently, investigators have emphasized the importance of determining whether the event is shocking to the individual or not, regardless of its form in order to define an event as traumatic, that is, able to produce symptoms of traumatic stress (intrusion, numbing, and arousal) [3]. Increasing evidence has documented the role of so-called low-magnitude events (eg, divorce, serious illness, and financial reverses) in determining post-traumatic stress reactions [4-6]. Conversely, several studies reported significant functional impairment and treatment seeking in a large number of victims who, although exposed to a DSM-IV-TR–qualified trauma, did not fulfill the symptom criteria (B, C, and/or D) [7-9].
The stressor Criterion-A1 and PTSD: A matter of opinion?
2009, Journal of Anxiety DisordersCitation Excerpt :Since definition of stress disorder in DSM-III (American Psychiatric Association, 1980), the effects of traumatic stress have been widely researched. However, the definition of the boundaries of the stressor “A1” criterion has emerged as one of the most controversial aspects of the diagnostic criteria (Breslau & Davis, 1987; March, 1993; Solomon & Canino, 1990; Spitzer, First, & Wakefield, 2007). For example, according to DSM-IV (American Psychiatric Association, 1994) to qualify as a traumatic event such an event should involve “actual or threatened death or serious injury, or threat to the physical integrity of self or others” (p. 427).
Research supported in part by a supplement to the Epidemiological Catchment Area Program (ECA) Cooperative Agreement No. U01 MH 33883 awarded to Principal Investigators Lee N. Robins and John E. Helzer of Washington University in St Louis, MO, for research performed with National Institute of Mental Health Principal Collaborators Darrel A. Regier, Ben Z. Locke, and Jack D. Burke, Jr.; the NIMH Project Officer was William Huber. Also supported by a supplement to Grant No. R01 MH36230 awarded to G.J.C. The opinions or assertions contained herein are the private ones of the authors, and are not to be considered as official or reflect the views of the NIMH or the Mental Health Secretariat (G.J.C.).
Presented at the 5th Annual Meeting of the Society for Traumatic Stress Studies, San Francisco, CA, October, 1989.
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