Skip to main content

Advertisement

Log in

Factors in neurological deterioration and role of surgical treatment in lumbosacral spinal lipoma

  • Original Paper
  • Published:
Child's Nervous System Aims and scope Submit manuscript

Abstract 

The purpose of this study was to determine factors that might be involved in neurological deterioration and the role of surgical treatment in patients with lumbosacral spinal lipoma. Pre- and postoperative courses of 34 patients were retrospectively analyzed. The age at surgery ranged from 1 month to 47 years. The records of preoperative neurological status indicated that older patients had more severe deficits, while all 8 asymptomatic patients were under 5 years of age. Motor deficits were noted in 9 patients, in 7 of whom the lipoma extended cranially beyond the L5 level. Transitional-type lipomas were accompanied by more severe deficits (asymptomatic 1, symptomatic 17) than other types (asymptomatic 7, symptomatic 9). Postoperative follow-up periods ranged from 5 months to 13 years. During these periods, 7 of the 8 asymptomatic patients remained neurologically intact. Nine of the 26 symptomatic patients improved. Age, extension of the lipoma in the spinal canal and type of lipoma will influence the preoperative neurological status of the patients. Early untethering surgery is recommended in patients with large lipomas extending beyond the L5 level.

This is a preview of subscription content, log in via an institution to check access.

Access this article

Price excludes VAT (USA)
Tax calculation will be finalised during checkout.

Instant access to the full article PDF.

Similar content being viewed by others

Author information

Authors and Affiliations

Authors

Additional information

Received: 24 August 1998 Revised: 19 July 1999

Rights and permissions

Reprints and permissions

About this article

Cite this article

Koyanagi, I., Iwasaki, Y., Hida, K. et al. Factors in neurological deterioration and role of surgical treatment in lumbosacral spinal lipoma. Child's Nerv Syst 16, 143–149 (2000). https://doi.org/10.1007/s003810050481

Download citation

  • Issue Date:

  • DOI: https://doi.org/10.1007/s003810050481

Navigation