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L-type calcium channel antagonist nifedipine reduces neurofilament restitution following traumatic optic nerve injury

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Mechanisms of Secondary Brain Damage from Trauma and Ischemia

Part of the book series: Acta Neurochirurgica Supplements ((NEUROCHIRURGICA,volume 89))

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Abstract

Background. The aim of the present study was to observe if the use of the L-type calcium channel antagonist nifedipine would offer advantages for the retinal ganglion cells and the restitution of the axonal cytoskeleton after optic nerve crush.

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Author information

Authors and Affiliations

  1. Institute of Anatomy Medical Faculty, Otto-von-Guericke University, Leipzigerstrasse 44, D-39120, Magdeburg, Germany

    Dr. J. Hanke

  2. Institute of Medical Psychology, Otto-von-Guericke University, Magdeburg, Germany

    B. A. Sabel

Authors
  1. Dr. J. Hanke
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  2. B. A. Sabel
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Editor information

Editors and Affiliations

  1. Institut für Chirurgische Forschung, Ludwig-Maximilians-Universität München, München, Germany

    A. Baethmann  & N. Plesnila  & 

  2. Neurochirurgische Klinik, Universität Würzburg, Würzburg, Germany

    J. Eriskat

  3. Neurochirurgische Klinik, Universität Freiburg, Freiburg, Germany

    J. Lehmberg

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© 2004 Springer-Verlag Wien

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Hanke, J., Sabel, B.A. (2004). L-type calcium channel antagonist nifedipine reduces neurofilament restitution following traumatic optic nerve injury. In: Baethmann, A., Eriskat, J., Lehmberg, J., Plesnila, N. (eds) Mechanisms of Secondary Brain Damage from Trauma and Ischemia. Acta Neurochirurgica Supplements, vol 89. Springer, Vienna. https://doi.org/10.1007/978-3-7091-0603-7_10

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  • DOI: https://doi.org/10.1007/978-3-7091-0603-7_10

  • Publisher Name: Springer, Vienna

  • Print ISBN: 978-3-7091-7206-3

  • Online ISBN: 978-3-7091-0603-7

  • eBook Packages: Springer Book Archive

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