Biochemical and Biophysical Research Communications
Regular ArticleEffects of Platelet-Activating Factor, Tumor Necrosis Factor, and Interleukin-1α on the Expression of Apolipoprotein M in HepG2 Cells
References (40)
- et al.
Lipoproteins and atherosclerosis
Baillieres Clin. Endocrinol. Metab.
(1987) High-density lipoprotein: A major risk factor for coronary atherosclerosis
Baillieres Clin. Endocrinol. Metab.
(1987)- et al.
ApoA-II maintains HDL levels in part by inhibition of hepatic lipase. Studies In apoA-II and hepatic lipase double knockout mice
J. Lipid. Res.
(1999) - et al.
Lipoprotein ApoC-II activation of lipoprotein lipase. Modulation by apolipoprotein A-IV
J. Biol. Chem.
(1990) - et al.
Overexpression of human apolipoprotein C-III in transgenic mice results in an accumulation of apolipoprotein B48 remnants that is corrected by excess apolipoprotein E
J. Biol. Chem.
(1994) - et al.
Apolipoprotein D
Biochim. Biophys. Acta
(2000) - et al.
Apolipoprotein L, a new human high density lipoprotein apolipoprotein expressed by the pancreas. Identification, cloning, characterization, and plasma distribution of apolipoprotein L
J. Biol. Chem.
(1997) - et al.
Plasma apolipoprotein L concentrations correlate with plasma triglycerides and cholesterol levels in normolipidemic, hyperlipidemic, and diabetic subjects
J. Lipid. Res.
(2000) - et al.
A novel human apolipoprotein (apoM)
J. Biol. Chem.
(1999) - et al.
Identification of platelet activating factor isolated from rabbit basophils as acetyl glyceryl ether phosphorylcholine
J. Biol. Chem.
(1980)
Regulatory role of phosphatidylinositol 3-kinase on TNF-alpha-induced cyclooxygenase 2 expression in colonic epithelial cells
Gastroenterology
Acth decreases the expression and secretion of apolipoprotein b in hepg2 cell cultures
J. Biol. Chem.
Single-step method of RNA isolation by acid guanidinium thiocyanate–phenol–chloroform extraction
Anal. Biochem.
Proposed lipocalin fold for apolipoprotein M based on bioinformatics and site-directed mutagenesis
FEBS. Lett.
Platelet-activating factor
J. Biol. Chem.
Inflammatory platelet-activating factor-like phospholipids in oxidized low density lipoproteins are fragmented alkyl phosphatidylcholines
J. Biol. Chem.
Oxidized LDL contains inflammatory PAF-like phospholipids
Trends Cardiovasc. Med.
N-linked glycosylation of macrophage-derived PAF-AH is a major determinant of enzyme association with plasma HDL
J. Lipid Res.
Human plasma platelet-activating factor acetylhydrolase. Oxidatively fragmented phospholipids as substrates
J. Biol. Chem.
Cited by (49)
The apoM-S1P axis in hepatic diseases
2020, Clinica Chimica ActaCitation Excerpt :Genetically, human APOM is situated on chromosome 6p21.3 in the major histocompatibility complex III, near regions rich in innate immunity and inflammation-related genes. Studies have verified that APOM can be regulated by several inflammatory factors, including platelet-activating factor, lipopolysaccharide and other factors [7–12]. S1P is verified only in apoM-containing HDL, and plasma levels of S1P in apoM-knockout mice are reduced by 46%, whereas they are increased in apoM transgenic mice compared to WT mice [13,14], indicating the important role of apoM in maintaining and regulating S1P levels.
Apolipoprotein M
2015, Clinica Chimica ActaCitation Excerpt :Specific stimuli, such as zymosan, turpentine and LPS, as well as TNF-α and IL-1, but not IL-6 correlated inversely with apoM transcription levels, the effects of which were mediated by cytokines. However, in another study, IL-1 and TNF-α did not influence apoM levels [69]. The underlying reasons for this discrepancy warrant further investigation.
Palmitic acid suppresses apolipoprotein M gene expression via the pathway of PPAR<inf>β/δ</inf> in HepG2 cells
2014, Biochemical and Biophysical Research CommunicationsUrinary apolipoprotein M could be used as a biomarker of acute renal injury: An ischemia-reperfusion injury model of kidney in rat
2013, Transplantation ProceedingsCitation Excerpt :It has recently been found in human colorectal tissues including cancers, cancer-adjacent normal tissues, polyps, and normal mucosa as well as in inflammatory mucosa.11 Several cytokines, including platelet activating factor (PAF), transforming growth factor-beta (TGF-β), epidermal growth factor (EGF), hepatocyte growth factor (HGE), and leptin, are known to regulate hepatic apoM expression in vivo and/or in vitro.12–16 Our previous study demonstrated hepatic apoM expression to be significantly influenced by hepatic IRI, suggesting that apoM has certain characteristics of an acute-phase reactive protein.
ABCA1 upregulating apolipoproein M expression mediates via the RXR/LXR pathway in HepG2 cells
2012, Biochemical and Biophysical Research Communications
- 1
To whom correspondence and reprint requests should be addressed. Fax: +46 –46 130064. E-mail: [email protected].